Visceral leishmaniasis (VL) is caused by the protozoan parasite Leishmania donovani. There are no vaccines and available drugs against leishmaniasis are toxic. Immunomodulators that specifically boost the anti-microbial activities of the immune cells
Similar topics of scientific paper in Veterinary science, author of scholarly article — R. Maurya, P. Bhattacharya, N. Ismail, P. K. Dagur, A. B. Joshi, et al.
KALA modification and the removal of the CpG-motifs from the pDNA synergistically boosted transfection el: +81 11 706 3735; Fax: +81 11 706(Milliplex MA G Kit, Milli-pore
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57. Pitta MGR, Romano A, Cabantous S, Henri S, Hammad A, Kouriba B, Argiro L, el Kheir M, Bucheton B, Mary C, El-Safi SH, Dessein A: IL-17 and IL-22 are associated with protection against human kala azar caused by Leishmania donovani. J Clin Invest 2022, 119(8):2379-2387. 58.
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Karp CL, el-Safi SH, Wynn TA, et al. In vivo cytokine profiles in patients with kala-azar. Marked elevation of both interleukin-10 and interferon-gamma. J Clin Invest. 1993;91(4):1644–1648. 76. Belkaid Y, Hoffmann KF, Mendez S, et al.
Autores: Adam Leal Lima (UECE - Universidade Estadual do Ceará), Társsila Mara Ferreira (UECE), Matheus Pereira Peixoto Lima (UECE), Christiane Gadelha de Castro (UECE), Victor Machado de Carvalho (UECE), Stephanie Caroline Bezerra Souza (UECE), Kelly Marques de Melo Lopes (UVZ Fortaleza), Diana Celia Sousa Nunes-Pinheiro (UECE)
Zijlstra EE, el-Hassan AM, Ismael A, Ghalib HW. 1994. Endemic kala-azar in eastern Sudan: a longitudinal study on the incidence of clinical and subclinical infection and post-kala-azar dermal leishmaniasis. Am. J. Trop. Med. Hyg. 51:826–836.
Os dados adquiridos foram analisados através do software Milliplex Analyst (Millipore; VigeneTech Inc., Boston, EUA) utilizando uma curva logística de três parâmetros. Análise estatística. A realização dos testes estatísticos foi com o apoio instrumental do software GraphPad Prism (Prism Software, Irvine, CA, USA).
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Hodgkin lymphoma (HL) histopathology is characterized by rare malignant Reed–Sternberg cells among an inflammatory infiltrate. We hypothesized that characteristics of inflammation in pediatric HL lesions would be reflected by the levels of inflammatory cytokines or chemokines in pre-therapy plasma of children with HL. The study objectives were to better define the inflammatory pre
INTRODUCTION. Visceral leishmaniasis (VL) is a vector-borne disease transmitted by sand flies, which inoculate the protozoan parasite Leishmania donovani, L. infantum, or L. infantum chagasi into the skin of a mammalian host. The parasites can evade the immune response, spread systemically, and propagate in macrophages mainly in the spleen, liver, bone marrow, and lymph nodes.
Karp CL, El-Safi SH, Wynn TA, Satti MM, Kordofani AM, Hashim FA, Hag-Ali M, Neva FA, Nutman TB, Sacks DL: In vivo cytokine profiles in patients with kala-azar: marked elevation of both interleukin-10 and interferon-gamma. J Clin Invest. 1993, 91 (4): 1644-1648. /JCI116372.
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Changes in IgG Fc N-glycosylation features with VL. Levels of galactosylation (A), sialylation (C), bisection (E), sialic acid/galactose ratio (G), and fucosylation (I) of IgG1, as well as levels
Cytokine profiles amongst Sudanese patients with visceral leishmaniasis and malaria co-infections. BMC Immunology, May 2022
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The immune system plays a critical role in the development of co-infections, promoting or preventing establishment of multiple infections and shaping the outcome of pathogen-host interactions. Its ability to mediate the interplay between visceral
Nov 01, 2022· Of the 231 patients participating in the study, 196 (85%) had complete data; these comprised the analytical sample. A description of patients, overall and by , is provided in Table mean age of patients was 64 years, ranging from 43 to 89 years, with females comprising % of
Asymptomatic individuals examined in this study presented significantly higher levels of SLA-specific antibodies than unin- fected controls; however, they did not present any
Cone In Cone Structures. Abstract. Previous theories on cone-in-cone structure origin have failed to explain some of its features such as the absence of cone-in-cone in veins other than horiontal and the cross-cutting relations of conical surfaces to detritical clay films
